Advanced Search
Display options
Filter resources
Text Availability
Article type
Publication date
Species
Language
Sex
Age
Showing 1 to 12 of 38 entries
Sorted by: Best Match Show Resources per page
Insights into the origin of DNA methylation differences between monozygotic twins discordant for schizophrenia.

Journal of molecular psychiatry

Melka MG, Castellani CA, O'Reilly R, Singh SM.
PMID: 26137221
J Mol Psychiatry. 2015 Jun 26;3(1):7. doi: 10.1186/s40303-015-0013-5. eCollection 2015.

BACKGROUND: DNA methylation differences between monozygotic twins discordant for schizophrenia have been previously reported. However, the origin of methylation differences between monozygotic twins discordant for schizophrenia is not clear. The findings here argue that all DNA methylation differences may...

Cite
Melka MG, Castellani CA, O'Reilly R, et al. Insights into the origin of DNA methylation differences between monozygotic twins discordant for schizophrenia. J Mol Psychiatry. 2015;3(1):7doi: 10.1186/s40303-015-0013-5.
Melka, M. G., Castellani, C. A., O'Reilly, R., & Singh, S. M. (2015). Insights into the origin of DNA methylation differences between monozygotic twins discordant for schizophrenia. Journal of molecular psychiatry, 3(1), 7. https://doi.org/10.1186/s40303-015-0013-5
Melka, Melkaye G, et al. "Insights into the origin of DNA methylation differences between monozygotic twins discordant for schizophrenia." Journal of molecular psychiatry vol. 3,1 (2015): 7. doi: https://doi.org/10.1186/s40303-015-0013-5
Melka MG, Castellani CA, O'Reilly R, Singh SM. Insights into the origin of DNA methylation differences between monozygotic twins discordant for schizophrenia. J Mol Psychiatry. 2015 Jun 26;3(1):7. doi: 10.1186/s40303-015-0013-5. eCollection 2015. PMID: 26137221; PMCID: PMC4487197.
Copy Download .nbib Format: NLM
The nucleus accumbens: a target for deep brain stimulation in resistant major depressive disorder.

Journal of molecular psychiatry

Nauczyciel C, Robic S, Dondaine T, Verin M, Robert G, Drapier D, Naudet F, Millet B.
PMID: 26019865
J Mol Psychiatry. 2013 Oct 23;1:17. doi: 10.1186/2049-9256-1-17. eCollection 2013.

OBJECTIVE: This review aimed to investigate the therapeutic potential of Deep Brain Stimulation (DBS) for treating resistant Major Depressive Disorder (MDD). We explored the role of Nucleus accumbens (Nac) as a target for treatment.METHOD: We made a systematic review...

Cite
Nauczyciel C, Robic S, Dondaine T, et al. The nucleus accumbens: a target for deep brain stimulation in resistant major depressive disorder. J Mol Psychiatry. 2013;1:17doi: 10.1186/2049-9256-1-17.
Nauczyciel, C., Robic, S., Dondaine, T., Verin, M., Robert, G., Drapier, D., Naudet, F., & Millet, B. (2013). The nucleus accumbens: a target for deep brain stimulation in resistant major depressive disorder. Journal of molecular psychiatry, 117. https://doi.org/10.1186/2049-9256-1-17
Nauczyciel, Cecilia, et al. "The nucleus accumbens: a target for deep brain stimulation in resistant major depressive disorder." Journal of molecular psychiatry vol. 1 (2013): 17. doi: https://doi.org/10.1186/2049-9256-1-17
Nauczyciel C, Robic S, Dondaine T, Verin M, Robert G, Drapier D, Naudet F, Millet B. The nucleus accumbens: a target for deep brain stimulation in resistant major depressive disorder. J Mol Psychiatry. 2013 Oct 23;1:17. doi: 10.1186/2049-9256-1-17. eCollection 2013. PMID: 26019865; PMCID: PMC4445576.
Copy Download .nbib Format: NLM
Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy.

Journal of molecular psychiatry

Nehls M.
PMID: 27429752
J Mol Psychiatry. 2016 Jul 15;4:3. doi: 10.1186/s40303-016-0018-8. eCollection 2016.

The aim of this review is to propose a Unified Theory of Alzheimer's disease (UTAD) that integrates all key behavioural, genetic and environmental risk factors in a causal chain of etiological and pathogenetic events. It is based on three...

Cite
Nehls M. Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy. J Mol Psychiatry. 2016;4:3doi: 10.1186/s40303-016-0018-8.
Nehls, M. (2016). Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy. Journal of molecular psychiatry, 43. https://doi.org/10.1186/s40303-016-0018-8
Nehls, Michael. "Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy." Journal of molecular psychiatry vol. 4 (2016): 3. doi: https://doi.org/10.1186/s40303-016-0018-8
Nehls M. Unified theory of Alzheimer's disease (UTAD): implications for prevention and curative therapy. J Mol Psychiatry. 2016 Jul 15;4:3. doi: 10.1186/s40303-016-0018-8. eCollection 2016. PMID: 27429752; PMCID: PMC4947325.
Copy Download .nbib Format: NLM
Preparation for teacher collaboration in inclusive classrooms - stress reduction for special education students via acceptance and commitment training: A controlled study.

Journal of molecular psychiatry

Pülschen S, Pülschen D.
PMID: 26421149
J Mol Psychiatry. 2015 Sep 28;3:8. doi: 10.1186/s40303-015-0015-3. eCollection 2015.

BACKGROUND: The education system in Germany is beginning to witness a sea change, lately, owing to the country's ratification of the United Nation's Convention on the Rights of Persons with Disabilities. The enactment is aiming at making provision for...

Cite
Pülschen S, Pülschen D. Preparation for teacher collaboration in inclusive classrooms - stress reduction for special education students via acceptance and commitment training: A controlled study. J Mol Psychiatry. 2015;3:8doi: 10.1186/s40303-015-0015-3.
Pülschen, S., & Pülschen, D. (2015). Preparation for teacher collaboration in inclusive classrooms - stress reduction for special education students via acceptance and commitment training: A controlled study. Journal of molecular psychiatry, 38. https://doi.org/10.1186/s40303-015-0015-3
Pülschen, Simone, and Pülschen, Dietrich. "Preparation for teacher collaboration in inclusive classrooms - stress reduction for special education students via acceptance and commitment training: A controlled study." Journal of molecular psychiatry vol. 3 (2015): 8. doi: https://doi.org/10.1186/s40303-015-0015-3
Pülschen S, Pülschen D. Preparation for teacher collaboration in inclusive classrooms - stress reduction for special education students via acceptance and commitment training: A controlled study. J Mol Psychiatry. 2015 Sep 28;3:8. doi: 10.1186/s40303-015-0015-3. eCollection 2015. PMID: 26421149; PMCID: PMC4587850.
Copy Download .nbib Format: NLM
αT-catenin in restricted brain cell types and its potential connection to autism.

Journal of molecular psychiatry

Folmsbee SS, Wilcox DR, Tyberghein K, De Bleser P, Tourtellotte WG, van Hengel J, van Roy F, Gottardi CJ.
PMID: 27330745
J Mol Psychiatry. 2016 Jun 21;4:2. doi: 10.1186/s40303-016-0017-9. eCollection 2016.

BACKGROUND: Recent genetic association studies have linked the cadherin-based adherens junction protein alpha-T-catenin (αT-cat, CTNNA3) with the development of autism. Where αT-cat is expressed in the brain, and how its loss could contribute to this disorder, are entirely unknown.METHODS:...

Cite
Folmsbee SS, Wilcox DR, Tyberghein K, et al. αT-catenin in restricted brain cell types and its potential connection to autism. J Mol Psychiatry. 2016;4:2doi: 10.1186/s40303-016-0017-9.
Folmsbee, S. S., Wilcox, D. R., Tyberghein, K., De Bleser, P., Tourtellotte, W. G., van Hengel, J., van Roy, F., & Gottardi, C. J. (2016). αT-catenin in restricted brain cell types and its potential connection to autism. Journal of molecular psychiatry, 42. https://doi.org/10.1186/s40303-016-0017-9
Folmsbee, Stephen Sai, et al. "αT-catenin in restricted brain cell types and its potential connection to autism." Journal of molecular psychiatry vol. 4 (2016): 2. doi: https://doi.org/10.1186/s40303-016-0017-9
Folmsbee SS, Wilcox DR, Tyberghein K, De Bleser P, Tourtellotte WG, van Hengel J, van Roy F, Gottardi CJ. αT-catenin in restricted brain cell types and its potential connection to autism. J Mol Psychiatry. 2016 Jun 21;4:2. doi: 10.1186/s40303-016-0017-9. eCollection 2016. PMID: 27330745; PMCID: PMC4915096.
Copy Download .nbib Format: NLM
Mitochondrial complex I and III gene mRNA levels in schizophrenia, and their relationship with clinical features.

Journal of molecular psychiatry

Akarsu S, Torun D, Bolu A, Erdem M, Kozan S, Ak M, Akar H, Uzun Ö.
PMID: 25713723
J Mol Psychiatry. 2014 Dec 10;2(1):6. doi: 10.1186/s40303-014-0006-9. eCollection 2014.

BACKGROUND: The etiology of schizophrenia is not precisely known; however, mitochondrial function and cerebral energy metabolism abnormalities were determined to be possible factors associated with the etiology of schizophrenia. Impaired mitochondrial function negatively affects neuronal plasticity, and can cause...

Cite
Akarsu S, Torun D, Bolu A, et al. Mitochondrial complex I and III gene mRNA levels in schizophrenia, and their relationship with clinical features. J Mol Psychiatry. 2014;2(1):6doi: 10.1186/s40303-014-0006-9.
Akarsu, S., Torun, D., Bolu, A., Erdem, M., Kozan, S., Ak, M., Akar, H., & Uzun, �. �. (2014). Mitochondrial complex I and III gene mRNA levels in schizophrenia, and their relationship with clinical features. Journal of molecular psychiatry, 2(1), 6. https://doi.org/10.1186/s40303-014-0006-9
Akarsu, Süleyman, et al. "Mitochondrial complex I and III gene mRNA levels in schizophrenia, and their relationship with clinical features." Journal of molecular psychiatry vol. 2,1 (2014): 6. doi: https://doi.org/10.1186/s40303-014-0006-9
Akarsu S, Torun D, Bolu A, Erdem M, Kozan S, Ak M, Akar H, Uzun Ö. Mitochondrial complex I and III gene mRNA levels in schizophrenia, and their relationship with clinical features. J Mol Psychiatry. 2014 Dec 10;2(1):6. doi: 10.1186/s40303-014-0006-9. eCollection 2014. PMID: 25713723; PMCID: PMC4307627.
Copy Download .nbib Format: NLM
Journal of molecular psychiatry.

Journal of molecular psychiatry

Thome J.
PMID: 25408894
J Mol Psychiatry. 2013 Apr 23;1(1):1. doi: 10.1186/2049-9256-1-1. eCollection 2013.

No abstract available.

Cite
Thome J. Journal of molecular psychiatry. J Mol Psychiatry. 2013;1(1):1doi: 10.1186/2049-9256-1-1.
Thome, J. (2013). Journal of molecular psychiatry. Journal of molecular psychiatry, 1(1), 1. https://doi.org/10.1186/2049-9256-1-1
Thome, Johannes. "Journal of molecular psychiatry." Journal of molecular psychiatry vol. 1,1 (2013): 1. doi: https://doi.org/10.1186/2049-9256-1-1
Thome J. Journal of molecular psychiatry. J Mol Psychiatry. 2013 Apr 23;1(1):1. doi: 10.1186/2049-9256-1-1. eCollection 2013. PMID: 25408894; PMCID: PMC4223887.
Copy Download .nbib Format: NLM
Circadian and behavioural responses to shift work-like schedules of light/dark in the mouse.

Journal of molecular psychiatry

McGowan NM, Coogan AN.
PMID: 25408900
J Mol Psychiatry. 2013 May 29;1(1):7. doi: 10.1186/2049-9256-1-7. eCollection 2013.

BACKGROUND: Disruption of circadian rhythms is associated with several deleterious health consequences and cognitive impairment. It is estimated that as many as one in five workers are exposed to this risk factor due to experiencing some degree of chronodisruption...

Cite
McGowan NM, Coogan AN. Circadian and behavioural responses to shift work-like schedules of light/dark in the mouse. J Mol Psychiatry. 2013;1(1):7doi: 10.1186/2049-9256-1-7.
McGowan, N. M., & Coogan, A. N. (2013). Circadian and behavioural responses to shift work-like schedules of light/dark in the mouse. Journal of molecular psychiatry, 1(1), 7. https://doi.org/10.1186/2049-9256-1-7
McGowan, Niall M, and Coogan, Andrew N. "Circadian and behavioural responses to shift work-like schedules of light/dark in the mouse." Journal of molecular psychiatry vol. 1,1 (2013): 7. doi: https://doi.org/10.1186/2049-9256-1-7
McGowan NM, Coogan AN. Circadian and behavioural responses to shift work-like schedules of light/dark in the mouse. J Mol Psychiatry. 2013 May 29;1(1):7. doi: 10.1186/2049-9256-1-7. eCollection 2013. PMID: 25408900; PMCID: PMC4223889.
Copy Download .nbib Format: NLM
Correction: Methylphenidate enhances neural stem cell differentiation.

Journal of molecular psychiatry

Bartl J, Mori T, Riederer P, Ozawa H, Grünblatt E.
PMID: 25408903
J Mol Psychiatry. 2013 Jun 10;1(1):10. doi: 10.1186/2049-9256-1-10. eCollection 2013.

No abstract available.

Cite
Bartl J, Mori T, Riederer P, et al. Correction: Methylphenidate enhances neural stem cell differentiation. J Mol Psychiatry. 2013;1(1):10doi: 10.1186/2049-9256-1-10.
Bartl, J., Mori, T., Riederer, P., Ozawa, H., & Grünblatt, E. (2013). Correction: Methylphenidate enhances neural stem cell differentiation. Journal of molecular psychiatry, 1(1), 10. https://doi.org/10.1186/2049-9256-1-10
Bartl, Jasmin, et al. "Correction: Methylphenidate enhances neural stem cell differentiation." Journal of molecular psychiatry vol. 1,1 (2013): 10. doi: https://doi.org/10.1186/2049-9256-1-10
Bartl J, Mori T, Riederer P, Ozawa H, Grünblatt E. Correction: Methylphenidate enhances neural stem cell differentiation. J Mol Psychiatry. 2013 Jun 10;1(1):10. doi: 10.1186/2049-9256-1-10. eCollection 2013. PMID: 25408903; PMCID: PMC4223885.
Copy Download .nbib Format: NLM
Genetics and epigenetics of alcohol dependence.

Journal of molecular psychiatry

Nieratschker V, Batra A, Fallgatter AJ.
PMID: 25408904
J Mol Psychiatry. 2013 Jul 11;1(1):11. doi: 10.1186/2049-9256-1-11. eCollection 2013.

Alcohol dependence is a severe and common disorder associated with high morbidity and mortality rates. Genetic as well as environmental factors are known to modulate susceptibility to alcohol dependence. There is a growing body of evidence suggesting that this...

Cite
Nieratschker V, Batra A, Fallgatter AJ. Genetics and epigenetics of alcohol dependence. J Mol Psychiatry. 2013;1(1):11doi: 10.1186/2049-9256-1-11.
Nieratschker, V., Batra, A., & Fallgatter, A. J. (2013). Genetics and epigenetics of alcohol dependence. Journal of molecular psychiatry, 1(1), 11. https://doi.org/10.1186/2049-9256-1-11
Nieratschker, Vanessa, et al. "Genetics and epigenetics of alcohol dependence." Journal of molecular psychiatry vol. 1,1 (2013): 11. doi: https://doi.org/10.1186/2049-9256-1-11
Nieratschker V, Batra A, Fallgatter AJ. Genetics and epigenetics of alcohol dependence. J Mol Psychiatry. 2013 Jul 11;1(1):11. doi: 10.1186/2049-9256-1-11. eCollection 2013. PMID: 25408904; PMCID: PMC4223883.
Copy Download .nbib Format: NLM
Neuroregeneration and dementia: new treatment options.

Journal of molecular psychiatry

Mitran SI, Catalin B, Sfredel V, Balseanu TA.
PMID: 25408905
J Mol Psychiatry. 2013 Jul 17;1(1):12. doi: 10.1186/2049-9256-1-12. eCollection 2013.

In the last years, physiological aging became a general concept that includes all the changes that occur in organism with old age. It is obvious now, that in developing and developed countries, new health problems concerning older population appear....

Cite
Mitran SI, Catalin B, Sfredel V, et al. Neuroregeneration and dementia: new treatment options. J Mol Psychiatry. 2013;1(1):12doi: 10.1186/2049-9256-1-12.
Mitran, S. I., Catalin, B., Sfredel, V., & Balseanu, T. A. (2013). Neuroregeneration and dementia: new treatment options. Journal of molecular psychiatry, 1(1), 12. https://doi.org/10.1186/2049-9256-1-12
Mitran, Smaranda Ioana, et al. "Neuroregeneration and dementia: new treatment options." Journal of molecular psychiatry vol. 1,1 (2013): 12. doi: https://doi.org/10.1186/2049-9256-1-12
Mitran SI, Catalin B, Sfredel V, Balseanu TA. Neuroregeneration and dementia: new treatment options. J Mol Psychiatry. 2013 Jul 17;1(1):12. doi: 10.1186/2049-9256-1-12. eCollection 2013. PMID: 25408905; PMCID: PMC4223882.
Copy Download .nbib Format: NLM
Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity.

Journal of molecular psychiatry

Şovrea AS, Boşca AB.
PMID: 26019866
J Mol Psychiatry. 2013 Oct 24;1:18. doi: 10.1186/2049-9256-1-18. eCollection 2013.

The goal of this review is to integrate - in its two parts - the considerable amount of information that has accumulated during these recent years over the morphology, biology and functions of astrocytes - first part - and...

Cite
Şovrea AS, Boşca AB. Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity. J Mol Psychiatry. 2013;1:18doi: 10.1186/2049-9256-1-18.
Şovrea, A. S., & Boşca, A. B. (2013). Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity. Journal of molecular psychiatry, 118. https://doi.org/10.1186/2049-9256-1-18
Şovrea, Alina Simona, and Boşca, Adina Bianca. "Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity." Journal of molecular psychiatry vol. 1 (2013): 18. doi: https://doi.org/10.1186/2049-9256-1-18
Şovrea AS, Boşca AB. Astrocytes reassessment - an evolving concept part one: embryology, biology, morphology and reactivity. J Mol Psychiatry. 2013 Oct 24;1:18. doi: 10.1186/2049-9256-1-18. eCollection 2013. PMID: 26019866; PMCID: PMC4445578.
Copy Download .nbib Format: NLM
Showing 1 to 12 of 38 entries